Wednesday, May 27, 2009

standard for so0cA

Slm.. sbb tak leh post kat blog kat sini je la.. ini adelah fwd email. mula2 ingtkn ramai yg dah dpt, tp td kat group ramai yg kate tak dpt lg..hope ade someone yg akan post ke blog batch..thanx. gud luck to all! tp ni lebih kurg je dgn info lain, tp still tak sedap hati if ade org yg tak dpt.Pape pun, ini cume guidelineYg penting, tawakkal. Semoga semua dipermudahkan. AMIN :)


SoOCA is objective student oral case analysis.
The objective of conducting SOOCA is assessing the comprehensive concept of a case.
The students are expected to analyze the case by explaining problems and its basic mechanism; rationale diagnosis, and therapy applying underlying basic sciences. (Including BHP, PHOP and CRP for batch 2008)

Items should be explained:

Start with describing mind mapping of the case (describing the correlations of one problem/condition to another: cause-effect relationships); checklist should accordance to any aspects that revealed from the mind mapping.

1. General case review: (score 5)
a. Explain the correlation of the main problem (diagnosis) with other conditions of the patient including etiology and/risk factors and/predisposing factors.
- Bacterial meningitis as complication of otitis media perforates (in the case describing otitis media perforates and bacterial meningitis)
Ischemic stroke with age, hypertension and hyperlipidemia as the risk factors (or any conditions that are mentioned in the case). If the affected vascular or part of the brain is mentioned then the student should also analyzed it.
b. Basis/criteria diagnosis using clinical interpretation of problems; including ways to rule out differential diagnosis (only those are mentioned in the case), including basis in the classification or types or affected structure, for exp. affected artery or part of the brain in stroke.

2. Basic sciences involved (20-35)
Explain/describe basic sciences being disturbed or affected in pathogenesis and pathophysiology.
- Motoric pathway in paralysis
Blood pressure regulation in hypertension
- Vascularisation in disease that spreads hematogenous
- Acid-base balance regulation and histology of renal tubule in acidosisdue to CRF; acid-base balance regulation and urine production/GFR in acidosis due to prerenal ARF. Different pathomechanism of the same problem leading to different basic sciences expected to be explained.
- Characteristic causing agent regarding morphology, structure/s of microorganism that play role in pathogenicity and virulence)

3. Pathogenesis: (10-20)
Explain the mechanism result in the disease and alteration structures that is showed in supporting examination such as radiology, pathology anatomy and others (if any)

4. Pathophysiology: (20-30)
Explain each mechanism results in all signs and symptoms including those revealed in laboratory examination.

5. Management: (5-10)
a. The principles (the purpose/s and the benefits) of non-pharmacological management including medical rehabilitation, prevention, patient education and others.
b. The principles/rationale approach of pharmacological management including the purpose/reason on drug selection.

6. Complication: (5)
Explain the mechanism result in the complication (if it mentioned in the case)

7. Prognosis: (5)
The reason of either good or poor prognosis

8. BHP (5 for 1 SKS)

9. PHOP (5)

10. CRP (5)

11. Student Performance (10)

Note: point 8, 9 and 10 are not for batch 2005-2007

Sumber: dr. Ike R H

all the best guys!

Monday, May 25, 2009



*know that while im typing this post im having palpitations of the fear of so0o0ca which is less than 13 come*

ok,just putting some info so, that everybody'll know.1st,d pembahagian markah:
  • pharmacokinetic+pharmacodynamic of drugs 10
  • anatomy,physiology,histology 30
  • synopsis of case 5-10
  • pathophysiology of case 10-15
  • pathogenesis of case 10-15
  • disease/case (epid,etio n d list goes on and on) 20
  • performance 10 doesnt exactly add up to 100.dont ask purely as blank as u is the targeted cases.kisi-kisi.act i think smua org pn dh tahu.but it doesnt hurt to know again kan?so,again,d kisi-kisi:
  • burn
  • fracture
  • psoriasis
  • acne
  • cleft lip and palate
  • anemia
  • ITP
  • SLE
  • anaphylactic shock
  • allergic rhinitis ( kowt.=P)
  • AIDS
  • CAD
  • heart failure
  • arrhythmia
  • MI
  • RHD
  • Buerger's disease
ntah.xtau sgt act.byk sgt msg.korg study je la pa2 korg rs best.hehe~dh,aq kn g siapkan case review.huhu~well,best of luck guys+gals!strive whether just to survive or excel excellently!yeah~

t8 cr!

Saturday, May 09, 2009



alhamdulillah.QbookCVS dh hbs di-edit around noon today.lega~yeay~n skrg tgh tunggu yoyo dtg amk i was going through d Qs td,mcm2 comment terlintas lam,tmpt tbaik nk tls adlh d sini.hehe~so,here it goes:

  • there are too many questions left unexplained n unanswered.maybe sbb kali ni bt awal kot.n theres still much that we havent,mmndgkn korg pn dpt buku awal,spend some time utk cr jwpn k.sorry bg phk QteamCVS. =(
  • skit ja rsnya soalan yg sm.but i havent bc satu2 yet la.just skali lalu,mmg mcm xdk yg sm.huhu~learn more,xlh nk agk jgk wether MDE CVS mcm MDE xslh berharap.hehe~
  • apa lg ek.dr segi allignment smua aq dh did d best.pnt tau.240 pg.soalan by soalan aq edit.line by line.word for word.hoho~n thankz samantha for doing a good job on intro pg.hehe~
  • hmmn~rs mcm tu ja.oh, 1 lg.theres a few members yg just ltk reference n pg n,if u guys tgh study soalan yg mcm tu,get ur books ready k.reference plg byk Lilly's.then Braunwald,lecture notes, lab manual n off course wiki2.hehe~
  • i wanna thankz QteamCVS for ur commitment.trima kasih byk2.Qteam lps ni sila join lg.hehe~n special thankz to fiza,da yg supply soalan hard copy n also soft copy from Qteam DMS-QteamHIS n,korg just jwb je xyh type.n also greenYa n noriz n dira for typing d twinning's Qs.thankz all!
thats all.if there's anythg else ill post lagi.hehe~well,loads n loads of luck for our exams!n jgn lp keep ur health check k.again,goodluck!

t8 cr!

Thursday, May 07, 2009


  • click for larger image.
  • strive!pray!goodluck!
t8 cr!